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Shanghai and Hong Kong researchers unlocks key heart repair mechanism_我的网站

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一 |     (ECNS) -- Zangnan, the southern part of the Xizang autonomous region, is China's territory, and China does not recognize the so-called "Arunachal Pradesh" illegally set up by India, said Foreign Ministry Spokesperson Guo Jiakun on Monday.    Guo made the remarks in response to questions about India's reported announcement on Friday of the "standard names" of 27 locations in "Arunachal Pradesh."    India's Ministry of Home Affairs said that identifying them formally on the map is aimed at "facilitating their accurate recognition and better awareness among the public at large."    "India's move of attempting to replace names long been used by China is illegal, null and void. Still less will it change the fact that Zangnan region belongs to China," Guo said.                  

Foreign Ministry Spokesperson Guo Jiakun. (Photo from fmprc.gov.cn)
         (By Gong Weiwei)                        。    (ECNS) -- A joint research team from Shanghai and Hong Kong published a landmark study in the journal Science on Friday, revealing the mechanisms behind the heart's innate emergency blood-supply pathway. The discovery could lead to game-changing treatments for acute myocardial infarction.    The 10-year collaborative project, jointly led by researcher Zhou Bin of the Center for Excellence in Molecular Cell Science at the Chinese Academy of Sciences (CAS) and Professor Kathy Lui of the Chinese University of Hong Kong, found that when major cardiac arteries are blocked during a heart attack, tiny capillaries – previously regarded as mere narrow “side paths” – are reprogrammed to remodel themselves into functional collateral arteries.    These spontaneous new vessels act as natural bypass channels, rerouting blood around blocked sections to rescue damaged heart tissue. The finding overturns the longheld belief that collateral arteries can only arise from preexisting arterial structures.    The breakthrough paves the way for targeted therapies to stimulate the heart to grow its own life-saving networks, offering a promising new treatment direction for all types of ischemic heart disease.    (By Intern Lin Qiaochu, Zhang Dongfang)                    。

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